SERUM PHOSPHORUS
Method: UV Molybdate / Photometric Method
| Parameter | Result | Units | Reference Range | Status |
|---|---|---|---|---|
| Serum Phosphorus | 3.8 | mg/dL | 2.5 – 4.5 | Normal |
Phosphorus plays an important role in bone mineralization, energy metabolism, cell membrane integrity, and acid-base balance. Serum phosphorus levels are closely regulated by the kidneys, parathyroid hormone, and vitamin D.
• Serum phosphorus level is within normal physiological limits.
• No evidence of hypophosphatemia or hyperphosphatemia.
• Clinical correlation advised if renal, endocrine, or metabolic disorders are suspected.
Note: Reference ranges may vary depending on age, diet, renal status, and laboratory methodology. Clinical correlation is recommended. This report is not valid for medico-legal purposes.
Method: UV Molybdate / Photometric Method
| Parameter | Result | Units | Reference Range | Status |
|---|---|---|---|---|
| Serum Phosphorus | 1.9 | mg/dL | 2.5 – 4.5 | Low |
Phosphorus is essential for bone formation, cellular energy production, muscle and nerve function, and maintenance of acid-base balance. Low serum phosphorus levels (hypophosphatemia) may occur due to poor dietary intake, malabsorption, vitamin D deficiency, alcoholism, hyperparathyroidism, diabetic ketoacidosis recovery, or renal phosphate loss.
• Serum phosphorus level is below the normal reference range.
• Findings are suggestive of hypophosphatemia.
• Clinical correlation is advised with nutritional status, renal function,
vitamin D levels, and parathyroid hormone status.
• Severe or persistent hypophosphatemia may be associated with muscle weakness,
bone pain, fatigue, or metabolic disturbances.
Note: Reference ranges may vary depending on age, diet, renal status, and laboratory methodology. Clinical correlation is recommended. This report is not valid for medico-legal purposes.
• Poor dietary intake or malnutrition
• Vitamin D deficiency
• Malabsorption syndromes / chronic diarrhea
• Hyperparathyroidism
• Chronic alcoholism
• Diabetic ketoacidosis recovery phase
• Renal phosphate wasting disorders
• Prolonged antacid use (aluminum/magnesium containing)
• Severe burns or sepsis
• Refeeding syndrome after prolonged starvation
Method: UV Molybdate / Photometric Method
| Parameter | Result | Units | Reference Range | Status |
|---|---|---|---|---|
| Serum Phosphorus | 5.8 | mg/dL | 2.5 – 4.5 | High |
Phosphorus plays an important role in bone mineralization, cellular energy production, muscle function, and acid-base balance. Elevated serum phosphorus levels (hyperphosphatemia) may result from impaired renal excretion, endocrine disorders, excessive phosphate intake, or cellular breakdown. Persistent elevation may contribute to soft tissue and vascular calcification, especially in patients with chronic kidney disease.
• Serum phosphorus level is above the normal reference range.
• Findings are suggestive of hyperphosphatemia.
• Correlation with renal function tests, calcium levels, parathyroid hormone,
and vitamin D status is advised.
• Persistent hyperphosphatemia may require further evaluation to identify
underlying renal, endocrine, or metabolic causes.
Note: Reference ranges may vary depending on age, diet, renal status, and laboratory methodology. Clinical correlation is recommended. This report is not valid for medico-legal purposes.
• Chronic kidney disease / renal failure
• Hypoparathyroidism
• Excessive vitamin D therapy or phosphate intake
• Tumor lysis syndrome or rhabdomyolysis
• Metabolic or diabetic ketoacidosis recovery phase
• Hemolysis or severe tissue breakdown
• Certain medications or phosphate-containing laxatives/enemas








